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MDSCs exert their immunosuppressive effects by inhibiting the function of T cells, B cells, natural killer cells, and other myeloid lineages, thereby dampening immune responses and facilitating tumor progression through the suppression of antitumor immune responses ( Recent studies have elucidated that the immunosuppressive function of MDSCs is intricately regulated by a myriad of complex metabolic pathways, with hypoxia playing a pivotal role in this regulation ( Furthermore, HIF-1α exerts profound effects on the recruitment, activation, and immunosuppressive functions of MDSCs