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Targeting mitochondrial transporters and metabolic reprogramming for disease treatment | Journal of Translational Medicine | Springer Nature Link
PMID: 278 PaikSShakSTangGKimCBakerJCroninMet al
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(242) found in their research that in hereditary leiomyomatosis and renal cell cancer cells, due to the inactivation of FH, GPX4 is succinated, leading to a decrease in its activity
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