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This is now a way, instead of that top-down approach to the regulation of fetal growth that I talked about, where it's sort of maternal adaptations to pregnancy, nutrients, hormones, then getting across the placenta, this is now a way that a fetal metabolism is compromised, or fetal metabolism is in such a state as to result in high amino acids, as well as the other things that might stimulate glucagon secretion are low oxygen, high catecholamines, like these sort of markers of stress or an inability to use amino acids

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