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FIGURE 5 Epigallocatechin-3-gallate nanoparticles inhibit the necroptosis rather than apoptosis in 2,4-dinitrochlorobenzene-induced atopic dermatitis To further explore whether inflammation triggers necroptosis after AD, the protein expression of RIP1, RIP3, and MLKL were measured (Figure 6)
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Disulfide bonds are relatively stable under physiological conditions but undergo selective cleavage in the tumor microenvironment (TME), minimizing premature drug release [53]
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