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Gene mutation or deficiency, chemotherapy, radiation or environmental toxicants can decrease glutathione peroxidase activity, increase NADPH oxidase subunits activity and induce the high level of ROS, leading to lipids/proteins peroxidation, endoplasmic reticulum stress, mitochondrial dysfunction, apoptosis, autophagy, ferroptosis, pyroptosis, necrosis, which ultimately lead to follicular atresia and premature ovarian insufficiency

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Inflammation is a primary contributor to the development of neurological disorders, as evidenced by the heightened expression of inflammatory factors in the CNS