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Interestingly, a recent study has demonstrated that compounds activating 5-HT2B receptors or inhibiting 5-HT2A receptors could enhance phosphoinositide 3-kinase (PI3K) signaling transduction and AMPA GluR1 dependent synaptic plasticity, and restore learning in Fmr1 knockout mice, further linking 5-HT receptors to fragile X syndrome (Lim et al., 2014)

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As this occurs, the cytoplasm within the process is gradually squeezed back toward the glial cell body, leading to the thinning and tightening of the layers around the axon