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Upon entering the cell, RSV activates SIRT1, leading to the inhibition of RelA acetylation, and stimulates inhibitor protein-B (IkB) degradation, which in turn reduces NF-kB-induced TNF-α, IL-1, IL-6, matrix metalloproteinase (MMP) and COX-2 production [77]

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6) are, however, not mutually exclusive and both could be exploited in future drug developments against ALS