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In neurodegenerative disease models, the co-delivery of GLP-1 analogues and conformation-specific antibodies against A β or α-syn has achieved concurrent neuroprotection and proteopathy clearance, leading to sustained behavioral and histological improvements [437, 438]

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Owing to its redox and metal-chelating capabilities, PDA readily adsorbs silver ions and directly reduces them on the fiber surface without an external reducing agent